Advances in multiple Sclerosis and Experimental Demyelinating Diseases
"There is a need for a paradigm shift in our thinking about the pathogenesis of multiple sclerosis." Challenging Charcot’s hypothesis that inflammatory response is the primary contributor to demyelination, Dr. Rodriguez and colleagues take a fresh, bold look at the causes and possible trea...
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| Otros Autores: | |
| Formato: | Libro electrónico |
| Idioma: | Inglés |
| Publicado: |
Berlin, Heidelberg :
Springer Berlin Heidelberg
2008.
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| Colección: | Current Topics in Microbiology and Immunology ;
318. Springer eBooks. |
| Acceso en línea: | Conectar con la versión electrónica |
| Ver en Universidad de Navarra: | https://innopac.unav.es/record=b32691671*spi |
Tabla de Contenidos:
- Benign Multiple Sclerosis: A Distinct Clinical Entity with Therapeutic Implications
- Pathological Heterogeneity of Idiopathic Central Nervous System Inflammatory Demyelinating Disorders
- Multiple Sclerosis Genetics
- Imaging of Remyelination and Neuronal Health
- Immunological Aspects of Axon Injury in Multiple Sclerosis
- The Multiple Sclerosis Degradome: Enzymatic Cascades in Development and Progression of Central Nervous System Inflammatory Disease
- Genetic Analysis of CNS Remyelination
- Remyelination in Experimental Models of Toxin-Induced Demyelination
- Remyelination-Promoting Human IgMs: Developing a Therapeutic Reagent for Demyelinating Disease
- Neuroimaging of Demyelination and Remyelination Models
- Hormonal Influences in Multiple Sclerosis
- Statins and Demyelination
- Role of Uric Acid in Multiple Sclerosis
- Neuromyelitis Optica: Clinical Syndrome and the NMO-IgG Autoantibody Marker.
